Hypothalamus – connection between the CNS and endocrine system.
Pituitary – is considered the select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 . Two lobes select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 and select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 . Posterior releases select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 and select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 . Anterior releases select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 which stimulates the adrenal cortexes. select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 targets the thyroid. select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 and select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 targets the gonads. select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 targets all body cells.
Thyroid – select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 and select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 stimulate metabolism. Also thyroid select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 calcium levels.
Parathyroid – select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 hormone select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 calcium levels.
Thymus – select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 promoted maturation of select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 .
Pancreas – Alpha produce select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 and Beta cells produce select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 .
Adrenals – select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 increase blood glucose levels. select one ATCH MASTER GLAND GLUCOCORTICOID INCREASES T3 OXYTOCIN LH MINERALOCORTICOID POSTERIOR T LYMPHOCYTES FSH GH DECREASES INSULIN PTH GLUCAGON THYMOSIN ADH ANTERIOR TSH T4 salt and fluid balance along with sodium and potassium balance.
Gonads – produce estrogen, progesterone and testosterone.
The Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS system controls the body through Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS called Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS and consists of Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS glands. The Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS system exerts its control through nervous impulses. The Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS system tends to be localized and use ducts. The word Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS is Greek for “to set into motion” and are needed to maintain body Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS . Select one HORMONES EIGHT NERVOUS EXOCRINE HORMONE HOMEOSTASIS METABOLISM ENDOCRINE CHEMICAL MESSENGERS is the sum of cellular process that produce energy needed to grow and repair.
Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN is marked by inadequate insulin activity in the body. Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN is important to all cells but especially the Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN . Glucose is the only substance that brain cells can readily and efficiently use as an energy source. Insulin also allows the body to store energy as Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN , protein, and fat.
The disease is named by a Greek doctor who found that affected peoples urine attracted bees. Hence Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN translates to “to pass through” and Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN means “honey sweet.” Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN comes from the Greek work “to change.” The building process within a cell to build is Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN . Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN comes from Greek “Up.” Breakdown process is called Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN , cata- is Greek for “Down.” So Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN makes or releases energy and Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN requires energy to drive them. Insulin stimulates the Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN pathways. Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN stimulates catabolic action. When sugar levels are low the body turns to Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ADIPOSE ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN cell break down into free fatty acids. In the liver catabolism of fatty acids produce Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN . As this acid accumulates it mixes with acetone and B-hydroxybutric acid and when all 3 are combined makes Select one DIABETES MELLITUS CATABOLISM ANABOLIC BRAIN ANABOLISM METABOLISM CATABOLIC ACETOECETIC ACID ANA- MELLITUS GLUCOSE KETONE BODIES DIABETES GLUCOGON GLYCOGEN which is called ketosis.
Normal blood levels are between 80 to 120 mg/dl. The Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON cells in the pancreas create Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON and beta cells produce Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON . Hypoglycemia is often defined as less than 80 mg/dl and hyperglycemia is over 140 mg/dl unless after a meal. After glucose levels in the blood rise above 180 mg/dl sugar is eliminated in urine. When glucose spills into urine the Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON is higher in the kidney than blood. Water follows sugar because of Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON . The term Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON means glucose in urine.
Type 1 Diabetes also called Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON onset or termed Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON . Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON accounts for most diabetic deaths. It is not uncommon for Type 1 to reach 300 – 500 mg/dl resulting in; Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON (thirst), Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON (excessive urination), and Select one ALPHA TYPE 1 POLYURIA INSULIN JUVENILE POLYPHAGIA GLYCOSURIA POLYDIPSIA INSULIN-DEPENDENT OSMOTIC DIURESIS OSMOTIC PRESSURE GLUCAGON (ravenous appetite).
Type II Diabetes termed Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE occurs most commonly in Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE patients and as a result decreases the number of insulin receptors per cell. Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE of all diabetics are Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE . General treatment is diet and exercise, oral hypoglycemic agents, and finally insulin injections.
Hyperglycemia is termed Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE and hypoglycemia is termed Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE . Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE is an advanced case of hyperglycemia and is life-threatening. When the pH level get to 6.9 the patient is hours from death. The onset is 12 to 24 hours, excessive hunger and thirst with general malaise. Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE is a major compensatory mechanism to remove CO2. The breath is fruity or Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE – like smell. Low Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE levels can be found because of the diuresis leading to cardiac dysrhythmias or death. Management is look for Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE or insulin in refrigerator. Complete a rapid glucose test. Draw red-top tube of blood. Give 1 to 2 liters of saline. If blood glucose levels are not available give fluid and 25G D50. The additional sugar will not hurt them but if they are in hypoglycemia it may save them. If the patient is a know alcoholic consider giving Select one 90% ACETONE ALERT BRACELET DIABETIC COMA THIAMINE DIABETIC KETOACIDOSIS NON-INSULIN POTASSIUM TYPE II KUSSMAUL'S INSULIN SHOCK OBESE 100mg prior to sugar.
HYPERGLYCEMIC HYPEROSMOLAR NONKETOTIC COMA (HHNK)
Serious complication associated with Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES diabetes. Both insulin and glucagons activity are present. This condition occurs when: 1) Sustained hyperglycemia causes osmotic diuresis to produce dehydration. 2) Water intake is inadequate to replace fluids. Most common causes are; Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES , Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES , and certain drugs. This patient still has insulin working so the Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES are not present. 40 to 70 mortality. How to pick it out = slower onset than Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES , over days. Increase urination and thirst. Volume depletion. Dry skin, Confused. Treatment is same as Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES because it is hard to diagnose in field.
Hypoglycemia is a Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES medical emergency. It takes hours for the brain to switch from sugar metabolism to fat metabolism and the damage cannot be reversed while waiting. Signs include stumbles, rage, and bizarre behavior. If levels fall critically low Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES are common. Always consider Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES when faced with a patient with bizarre behavior. The onset is Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES . Care is if glucose is less than 60 mg/dl draw red top. 25 to 50G D50, If IV cannot be started give Select one SEIZURES SUDDEN TYPE II INFECTION GLUCOGON INSULIN SHOCK TRUE DKA DIALYSIS KETONES 0.5 to 1 mg IM.
HYPERTHYROIDISM is Select one PROLONGED INADEQUATE EXCESS LOW thyroid hormones in blood.
THYROTOXICOSIS is Select one PROLONGED INADEQUATE EXCESS LOW exposure to excessive thyroid hormones.
HYPOTHYROIDISM is Select one PROLONGED INADEQUATE EXCESS LOW thyroid levels.
MYXEDEMA is long term exposure to Select one 95% PROLONGED FEVER INADEQUATE THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW levels of thyroid hormones.
GRAVES’ DISEASE
Graves’ Disease accounts for over Select one 95% PROLONGED FEVER INADEQUATE THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW of Select one 95% PROLONGED FEVER INADEQUATE THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW and 15% have relatives with the disease. Graves’ is more common in Select one 95% PROLONGED FEVER INADEQUATE THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW . Onset is 20’s to 30’s. Autoimmune autobodies keep stimulating the thyroid to produce too much. Signs are agitation, insomnia, tachycardia, increase appetite, poor heat tolerance. Protrusion of the Select one 95% PROLONGED FEVER INADEQUATE EYE BALLS THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW and large goiter. Care is monitor cardiac activity. Beta blockers may help heart. Steroid like Select one 95% PROLONGED FEVER INADEQUATE THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW may quickly reduce T4 levels.
THYROTOXIC CRISIS also know as Select one 95% PROLONGED FEVER INADEQUATE THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW
True life-threatening and kills within Select one 95% PROLONGED FEVER INADEQUATE THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW hours. Most common when body is under stress from trauma or infection. The patient has inactive thyroid circulating but for some reason the inactive becomes active which overdoses the blood. Signs are increased activity of sympathetic system, high Select one 95% PROLONGED FEVER INADEQUATE THYROID STORM 48 THYROTOXICOSIS WOMEN DEXAMETHASONE EXCESS LOW 106, delirium, coma, tachycardia, vomit, diarrhea. Same as Graves is the care.
Hypothyroidism
Creates a Select one MYXEDEMA WEIGHT GAIN WEIGHT LOSS LOW MYXEDEMA COMA metabolic state. This results in poor handling of exercise and infection. Over time develops into Select one MYXEDEMA WEIGHT GAIN WEIGHT LOSS LOW MYXEDEMA COMA . Select one MYXEDEMA WEIGHT GAIN WEIGHT LOSS LOW MYXEDEMA COMA is a thickening of connective tissue in skin and other tissue organs of the body like the heart. Become hypothermic in nature, and go into a stuporous state called Select one MYXEDEMA WEIGHT GAIN WEIGHT LOSS LOW MYXEDEMA COMA . Sings include slowing mental status and function, decrease appetite with weight gain, constipation, bradycardias. Care is support ABC and cardiac. Limit fluids to not alter electrolyte balance.
Adrenal Gland
Select one CORTISOL SODIUM AUTOIMMUNE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS is called Select one CORTISOL SODIUM AUTOIMMUNE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS . Cushings syndrome is relatively common. Middle aged persons with women more than men. Excess Select one CORTISOL SODIUM AUTOIMMUNE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS which is a Select one CORTISOL SODIUM AUTOIMMUNE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS , can be caused by the Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS or adrenal cortex. Also people who are treated with steroid such as Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS . Early sign is Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS . Long term exposure to steroids is an antagonist to insulin so Select one DIABETES CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS can develop. Fat metabolism is altered so Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS is common. Sodium retention so they look puffy. Earliest sign is WEIGHT GAIN in the trunk and face and neck. A Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS develops. A Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS develops. Thinning of skin and women getting facial hair. Care is limited in field remember that atherosclerosis and hypertension can lead to heart problems and strokes. Use care when starting an IV.
Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS is caused from Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS . It is due to steroid destruction. This is an Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS disease like Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS . Infection and trauma can be a problem because the do not have proper steroid production. Addison’s causes major disturbances in water and electrolyte balance. Increase Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS excretion cause low volume but an increase in Select one CORTISOL SODIUM AUTOIMMUNE PREDNISONE ANTERIOR PITUITARY POTASSIUM BUFFALO HUMP HYPERADRENALISM GRAVES ADDISONS DISEASE ADRENAL INSUFFICIENCY MOON FACE CUSHINGS SYNDROME WEIGHT GAIN STEROID ATHEROSCLEROSIS to hyperkalemia is likely. One cause of Addison’s is use of steroids like prednisone. While on the drug the adrenal glands are suppressed. Signs include fatigue, weight loss, hyperpigmentation of skin. Care is aggressive fluid if needed, check sugar levels and treat, get 12 lead and ECG monitory for cardiac events.
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